Aug 9, 2013 · Exhaled NO (eNO) is an early, non-invasive biomarker for different types of acute lung injury. Its sensitivity is improved by normalizing eNO to exhaled CO 2 and breathing frequency. eNO appears to be a particularly sensitive indicator for acute airway injury. Inhalation treatment with a nitric oxide synthase inhibitor is mirrored by decreased eNO.

Aug 9, 2013 · Exhaled NO (eNO) is an early, non-invasive biomarker for different types of acute lung injury. Its sensitivity is improved by normalizing eNO to exhaled CO 2 and breathing frequency. eNO appears to be a particularly sensitive indicator for acute airway injury. Inhalation treatment with a nitric oxide synthase inhibitor is mirrored by decreased eNO.

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Nov 23, 2010 · The aim of this study was to determine whether or not AIV induces eNO dependent vasodilatation and to study its frequency and power dependence Methods: Male rats (±275 g) were instrumented for continuous monitoring of blood pressure, frequency of AIV and electrocardiogram.

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Conclusions: Inhaled ENO conferred protection superior to inhaled NO against hyperoxia-induced inflammation. ENO prevented hyperoxia impairments of lung compliance and postnatal alveolar development in newborn rats. Keywords: bronchopulmonary dysplasia; O-nitrosoethanol; S …

Feb 20, 1998 · In this study, we investigated the effect of acidosis on nitric oxide production in healthy anesthetized rats, and evaluated the relationship between acidosis, NO, hypotension, and organ damage.

The eNOS protein level, evaluated by Western blotting, was evident as an intense band in cardiac homogenates of non-diabetic and diabetic rats. The expression of mRNA for eNOS did not differ significantly between the two groups.

We found that both inhaled ENO and NO partly prevented hyperoxia-induced leukocyte influx in BALF in newborn rats. In contrast, 10 ppm ENO treatment was superior to 10 ppm NO at preventing hyperoxia-induced accumulation of MPO.

Rats were exposed to either NaCl (normal control) or 10 mg/kg PFOS for 30 days, and ENO2 protein expression was assessed by immunofluorescence staining. The results, as shown in Fig. 7 , revealed that ENO2 was widely expressed in the rat brain.